• 支援肝臟健康
• 支援身體自然產生排毒酵素的能力，以去除體內有害毒素
• 有助清除肝臟與血液裡的毒素
• 有助排除體內日積月累的毒素
• 提供強效的抗氧化保護，讓身體免於受到自由基帶來的負面影響
• 支援健康的免疫功能
• 促進不健康細胞的自然凋亡
• 有助維持健康的穀胱甘肽水平
• 支援正常的穀胱甘肽合成

*這些陳述未經過美國食品藥物管理局評估。
本產品無意作為診斷、治療或預防任何疾病之用。

哪些人應該購買淨腸排毒組合？

任何18歲或以上人士均可使用淨腸排毒組合內的產品，尤其是想要支援正常的肝臟解毒活性，幫助維持健康的穀胱甘肽濃度，提供強效的抗氧化保護，讓身體免於受到自由基帶來的負面影響。

什麼是穀胱甘肽？為何對排毒過程重要？

穀胱甘肽在排毒過程中進行兩項任務。首先，它能抑制肝臟在排毒第一階段中和毒素時產生的自由基。其次，在排毒的第二階段，它與有毒化學物質結合（如香菸煙霧、汽車尾氣和致命的多氯聯苯中的有害物質），使之安全地排出體外。

穀胱甘肽還能將小腸中的有害物質在其被吸收進入血液前排出。它還被認為可保護您的胃黏膜免受其產生大量酸的自由基影響。高濃度的穀胱甘肽有益於免疫系統，使身體產生更多的白血球以對抗感染和清除有害毒素。

淨腸排毒組合所含產品已被證實可幫助身體產生穀胱甘肽，而穀胱甘肽對酵素作用有舉足輕重的地位。人體需要酵素作用來適當的過濾及消除體內毒素。穀胱甘肽也被視為第二期酵素，當第一期酵素發揮作用過濾出毒素後，穀胱甘肽可使這些物質更具水溶性，從而透過膽汁或腎臟排出體外。

肝臟排毒與減重排毒有什麼區別？

很多人一聽到「排毒」就想到「減重」。雖然開始減重計畫的人士通常都會經歷一段時間的排毒，他們排毒的原因卻不相同。

建議每年做一到兩次減重排毒，每次通常持續3至7天，期間只吃某些食物，除此以外還使用一些產品，比如NutriClean®7天淨腸排毒組（結腸和肝臟淨化系統）來為消化道和肝臟清潔排毒，進而支援身體自然的恢復和淨化過程。

淨腸排毒組合可用於支援身體正常的排毒過程，沒有週期性排毒系統常見的限制。這包括持續清除自由基以清除體內堆積的毒素，提供強大而持久的抗氧化保護來防止毒素聚積，維護肝臟健康，進而幫助身體規律清潔而不是只在短期內清潔。

為何肝臟至關重要？

肝臟是人體內最重要的器官之一。它是消除許多不斷進入人體之非天然、有害物質的主力戰將。在自由基、有毒物質及化學物品到處充斥的現代生活中，維持肝臟健康不容忽視。肝臟的功能是在找出並消滅所有的有害物質，並將有害物質排除體外。為了維持健康，甚至可說是維持生命，人體需要肝臟規律地發揮這項功能。肝臟能分解在身體內部產生的複合物，如過多的荷爾蒙及其他由正常代謝功能產生的副產品。肝臟過濾並排除來自體外的物質如食物、食品防腐劑、添加的化學物質、污染物、致病的毒素及過量的酒精所帶來的損害。生活方式如遺傳、飲食、環境及壓力等我們能控制或不能控制的因素，會影響能否控制肝臟功能正常，也因此會影響肝臟過濾並消除有毒物質的能力。

Timeless Prescription®美安常青配方膠囊食品：

維生素A（β-胡蘿蔔素）(Vitamin A (Beta-Carotene))：5000國際單位
維生素A是一種最高等級的抗老化微量營養素，與維生素C、維生素E和礦物質硒的功效並駕齊驅，有助維持健康。多年來所進行的研究指出，β-胡蘿蔔素或在維持整體健康方面起重要作用。作為維生素A的前驅物，β-胡蘿蔔素也因此被稱為原維生素A化合物。含有β-胡蘿蔔素的食物或營養補充品可被轉化為維生素A，以維持肌膚健康、良好視力以及健康的免疫系統。

維生素C(Vitamin C)：200毫克
亦稱為抗壞血酸，維生素C有助維持肌膚的健康膠原蛋白，促進正常的療癒過程，促進健康的牙齒和骨骼，並支援免疫系統。維生素C是較為強效並廣為人知的抗氧化物之一。正如同將蘋果的切口暴露於空氣中會使其迅速變成褐色一樣，身體的細胞與氧氣接觸時也會受到影響，此過程被稱為氧化。氧化會造成肌膚以及身體其他器官與組織的老化。作為抗自由基成分，維生素C可幫助身體更長時間地保持年輕。

維生素E(Vitamin E)：30國際單位
維生素E可支援呼吸系統，亦可促進免疫系統的健康。維生素E可保護身體免受氧化傷害；氧化是一個正常老化過程，而氧氣會藉此破壞身體組織。維生素E可中和自由基。自由基是自然產生的不穩定分子，藉由取得電子以維持自身的平衡，卻會破壞體內的健康分子。當體內含有足夠的維生素E的時候，不穩定的自由基會從維生素E取得所需的電子，而不會影響體內的健康分子，進而促進組織健康。

硒(Selenium)：200微克
硒是維持長壽的健康方案中最重要的微量營養素之一，抗老化專家經常引用為抗氧化物，應將其列入抗老化清單中。硒和維生素E，也是另一個強效抗氧化物，均可協效運作；意即這兩者可互相促進彼此的健康效果。而且，硒可促進身體主要的抗氧化物穀胱甘肽過氧化物酶的生成。硒是一種重要的微量礦物質，與維生素E密切合作，以提供身體防禦自由基的抗氧化防護。硒以兩種方式保護身體抵抗自由基：可與蛋白質結合，產生對抗氧化酵素非常重要的硒蛋白；也支援身體產生天然的抗氧化物，穀胱甘肽。

山桑果(Bilberry)：25毫克
作為抗氧化物，山桑果的主要作用是其與維生素E的強效協力作用。在最基本的層面上，它可支援微血管的正常抵抗力並促進其不滲透性。山桑果花青素亦可支援視網膜細胞代謝與功能中的關鍵酵素運作。它還可以支援心臟健康。山桑果支援正常的血小板活性。

綠茶萃取物(Green Tea Extract)：300毫克
綠茶萃取物是一款可以促進健康的抗氧化物。它富含多酚，可以促進健康。

銀杏(Ginkgo Biloba)：50毫克
銀杏有助支援記憶力、大腦功能、情緒、腦部和末梢循環、以及供氧和血流。

牛奶薊(Milk Thistle)：50毫克
牛奶薊被用於促進肝臟健康，含有數種已知最強效的保護肝臟物質。可發揮抗氧化功能來防禦自由基，進而保護肝臟。支持新肝細胞的正常生成。

Curcumin Extreme™薑黃素配方：

薑黃萃取物(BCM-95®)(Curcumin (BCM-95®))：400毫克
科學家早已注意到印度香料薑黃的許多健康益處。薑黃是活性植物生化素薑黃的來源。目前已知薑黃萃取物的生物可用性偏低，需攝取高劑量方有益健康。與其他來源的薑黃萃取物相比，BCM-95®在血液中提供明顯更多的藥理上具有生物活性的薑黃素。這項新的傳輸系統可以帶來各種健康好處。

這是如何實現的？ 傳統的薑黃95%萃取物完全側重於其根莖部位。薑黃(Curcuma Longa)的生物活性物質含有「類薑黃素」，而薑黃素是最為重要的分子。經研究顯示，薑黃可帶來巨大的健康益處。雖然薑黃素是最重要的分子，但市售的一般薑黃95%萃取物的生物可利用性在血液中的吸收或持續性卻不是非常好。薑黃根莖中含有其他必需成分，而傳統的薑黃95%萃取物製造方法往往忽略這些成分。BCM-95®代表著薑黃根莖天然全成分。它是全天然的，並且經證實可提供協效運作所需的最佳生物可利用性。這種全新製造方法在生物可利用性方面提供極大的價值。

人們對抗氧化物的關注與日俱增，而且我們必須了解哪些營養素具有抗氧化性的成分，以及它們的相關資訊；薑黃素就是此類營養成分之一。薑黃素是取自香料薑黃的天然萃取物。薑黃素是從一種薑科植物薑黃(Curcuma Longa)中萃取而得。

雖然在傳統上，薑黃素可促進胃部和關節舒適，但它主要被用作抗氧化物。薑黃素的平衡免疫活動已透過多種機制得到證實，可維持體內正常的環氧化酶-2與核因子活化B細胞κ輕鏈增強子濃度。

薑黃素的神經保護特性是最為廣泛研究的。薑黃素被認定為可促進神經系統健康與認知功能的「強力候補」。薑黃素可越過血腦屏障，促進大腦澱粉樣β蛋白的正常攝取。隨著年齡增長，支援腦部記憶力與學習能力。薑黃素的另一個神經保護特性就是促進大腦中的穀胱甘肽、超氧化物歧化酶以及過氧化氫酶達到正常水平，有助維持神經組織的健康。

薑黃素支援二期肝臟排毒酵素的正常分泌，包括穀胱甘肽合成酵素、血紅素氧化酶和過氧化氫酶。肝臟在排毒過程中扮演多種角色：過濾血液以去除其中的大型毒素、合成並分泌富含膽固醇和其它脂溶性毒素的膽汁，並產生酵素分解身體不需要的化學物質。這個酶促過程通常分為一期和二期兩個步驟。它們可增強人體天然酵素的抗氧化防禦系統和功能，是功效強大的間接性抗氧化物。這些酵素可促進身體將有害化學物，例如重金屬、毒素和污染源，正常代謝為活性較低的分子。薑黃素已知能促進正常的肝臟組織修護。

綠花椰菜籽萃取物（6％蘿蔔硫素硫配糖體）(Broccoli Seed Extract (6% Sulphoraphane Glucosinolates))：167毫克
過去25年，一直有文獻記錄綠花椰菜及其他十字花科蔬菜的健康好處與保護功能。綠花椰菜籽萃取物含豐富蘿蔔硫素硫配糖體。蘿蔔硫素支援二期肝臟排毒酵素的正常分泌，包括穀胱甘肽合成酵素、血紅素氧化酶和過氧化氫酶。蘿蔔硫素可增強人體天然酵素的抗氧化防禦系統和功能，是功效強大的間接性抗氧化物。蘿蔔硫素可支援基因轉錄，是將遺傳資訊由脫氧核糖核酸(DNA)複製到RNA的過程，最終轉譯成特定的蛋白質形式。是以蘿蔔硫素有助於促進人體天然的防禦系統，維持更高的穀胱甘肽濃度。

穀胱甘肽是人體主要的抗氧化物。它是重要的化學物質，作為強效抗氧化物，可維護和保護腦部以及人體其他組織，使其免受自由基的損害。此外，穀胱甘肽亦可循環利用能夠減少自由基的維生素C及E。由於腸胃道降解造成穀胱甘肽無法透過口服完整吸收，因此補充蘿蔔硫素可能是增加內生穀胱甘肽濃度最有效的方法。

硒（硒甲硫胺酸）(Selenium (Selenomethionine))：100微克
硒是硒蛋白（例如穀胱甘肽過氧化物酶）的必要輔因子。硒甲硫胺酸會直接並入蛋白質，因為在傳訊RNA轉譯為蛋白質的過程中，硒甲硫胺酸無法從甲硫胺酸中分離。可作為硒的儲存形式，並且在蛋白質分解代謝時釋放。硒會累積在攝護腺，促進攝護腺健康。藉由促進輔助型T細胞的正常生長與發育，硒可支援免疫功能。

NutriClean®清肝配方的主要成分：

牛奶薊籽(Milk Thistle Seed)（標準化萃取水飛薊素含量為80%）

牛奶薊有清除自由基的功能，因此可助促進肝臟及膽囊的健康，並可刺激肝細胞的再生，防止肝細胞吸收毒素。當肝臟細胞遇上不同的毒素時，會自行產生強烈的自我保護反應。牛奶薊可幫助受損的肝臟組織再生，維護肝臟及膽汁膽固醇的健康，還可減少肝臟因氧氣供應不足所帶來的傷害。牛奶薊含有一種名為水飛薊素的複合物質，它是一種強大的抗氧化物，能清除體內有害的自由基。經發現，牛奶薊亦可修復腎臟功能，有益攝護腺健康。水飛薊素這個名稱，是黃酮木酚素這類活性化學物質的通稱，這種物質僅存在於牛奶薊籽中。水飛薊素，又稱為水飛薊賓，是黃酮木酚素的分離形式，對肝臟具有一種特殊的功能。它可藉著降低或預防因酒精、環境、細菌及毒藥等毒素造成的傷害，來維護肝臟的健康。初步證據顯示，水飛薊素可支援健康的血清轉氨酶水平以及肝臟健康。許多研究表明，水飛薊素可以有效淨化肝臟。

N-乙酰基-L-半胱氨酸(N-Acetyl-L-cysteine)

N-乙酰基-L-半胱氨酸（乙醯半胱胺酸）是從半胱胺酸這種含有硫磺的氨基酸內導出的物質。NAC可由人體自行製造，亦存在於食物中。乙醯半胱胺酸可以調解（與谷胺酸及甘胺酸一起）半胱氨酸轉化為穀胱甘肽的過程。穀胱甘肽是人體內一種主要的抗氧化物質。研究指出，乙醯半胱胺酸具有支援免疫系統、解除重金屬毒素、維護心臟健康、舒緩宿醉症狀及減低運動疲勞等功能。一般認為，乙醯半胱胺酸在體內的兩種主要作用均可作為它的建議好處。首先，乙醯半胱胺酸可快速代謝成細胞內穀胱甘肽。而穀胱甘肽(GTH)以及它所形成的酵素複合物便成為體內的還原劑及抗氧化物。穀胱甘肽可將體內的化學毒素緩解成較不具殺傷力的複合物質，並可排解體內的汞及鉛等重金屬毒素。穀胱甘肽同時具有幫助運送營養素至淋巴細胞及薄膜這兩個體內主要的免疫細胞的功能，它還能保護細胞黏膜。乙醯半胱胺酸則是被認為是提升細胞穀胱甘肽值的最佳來源。乙醯半胱胺酸可藉著將二硫鍵轉化為兩個巰基群組，來裂解二硫鍵。這個動作可分解肺部黏液中的黏蛋白，減低黏液的鏈長，並使黏液變薄。而且，乙醯半胱胺酸還被證實有助健康的血小板聚集，並可維護健康的脂蛋白值。

數種研究證實乙醯半胱胺酸在體內會轉化成穀胱甘肽。這些研究的評論顯示，口服乙醯半胱胺酸補充品能有效的提高肝臟、肺臟血漿及支氣管內的穀胱甘肽值。乙醯半胱胺酸同時可幫助降低疲勞感，並協助耗能運動時的肌肉收縮（很可能是由於體內氧化壓力值降低）。

蒲公英根(Dandelion Root)

蒲公英是前院草地上常見，極不易清除的頑固植物，但它其實是種極難得的營養食物。它的葉片含有大量的維生素A，以及一般含量的維生素D、C、多種維生素B群、鐵、矽、鎂、鋅及錳。蒲公英的英文名稱來自法文的dent de lion，也就是獅子的牙齒，指的便是它齒狀邊緣的葉片。蒲公英根已被證實有助肝臟、腎臟、膽囊及關節的健康。

蒲公英根被視為「肝臟的補藥」，據信它能夠以一種尚未被詳細解釋的方法來維護肝臟健康。蒲公英內的苦味成分有助膽囊內膽汁的健康分泌，並有助肝臟的膽汁流量。這種複合物之前被稱為蒲公英苦素，是蒲公英植物所獨有的。這些成分是桉烷內酯及大根香葉內酯類的倍半萜內酯。蒲公英的苦味被認為具有使緩慢的肝臟功能加速運作的功用。而增加膽汁流量也有助於健康的體內脂肪（包括膽固醇）代謝。蒲公英根也像其它具有苦味的香草植物一般，被用來幫助恢復食慾及正常的消化系統。

大蒜(Garlic Bulb)

中國人早在西元前510年左右，就開始使用大蒜。法國微生物學家Louis Pasteur在1858年，便研究大蒜的抗菌功效。現今大蒜廣為世人使用，因為它對心血管系統，起了很好的保護作用。大蒜專家們都認為，大蒜可維持體內健康的膽固醇值，好對抗日漸硬化的血管。大蒜也可維護人體正常的三酸甘油酯值。大蒜本身也含有一些抗氧化物。

山楂果萃取物(Hawthorn Berry Extract)

山楂果，或稱西洋山楂，是一種長著白花或粉紅花的多刺灌木，它所結的果實，很像縮小版的蘋果。這種灌木原生長於溫帶氣候的亞洲、歐洲以及北美州東部。中國人將山楂果用於幫助消化及做為食品已有數百年的歷史。早在文藝復興時代，歐洲人便使用山楂果來幫助消化。西元1800年代晚期，歐州的醫生將山楂果運用於維護心臟健康上。近年來，山楂果被用來做為強心劑（強化心肌並促進更有力的收縮）。山楂果的主要益處似乎是促進心臟健康。山楂果含有一群被稱為類黃酮的化學物質，是此植物的主要成分，被認為可對心臟健康產生數種幫助。首先，山楂果萃取物有助維持心血管健康，改善血液流動。其次，山楂果有助維持正常血壓。作為抗氧化物，山楂果有助維持血管壁的膠原蛋白基質的健康，以維護血管彈性。

五味子果實及種籽萃取物(Schizandra Chinensis (Fruit and Seed Extract))

自古以來，中國人便大量使用五味子這種被視為具養生及增加活力功效的草藥。五味子是木蘭科植物的一種，來自藤蔓般的植物所結的紅莓果上。目前全球大約有25種不同的五味子，其中僅有一種是生長於美國北卡羅萊納州以及附近幾個州的林木區，其它全部生長在亞洲。五味子的功效除了傳統上用來增強能量以及消除壓力造成的倦怠感外，它還能增強性能力、提升心智功能、改善膚質以及治療盜汗。五味子還可促進肝臟及腎臟健康，提高攝氧量及能量，改善視力以及增強心智功能。

五味子是適應原家族的一分子，內含據信可促進體內平衡的物質。五味子含有木脂素，是五味子莓果種籽的一種濃縮成分，有提升免疫系統功能的作用，還有護肝臟以及增強身體抵抗壓力的能力。

皺葉酸模根(Yellow Dock Root)

皺葉酸模是蕎麥家族的一員，原本生長在北歐及亞洲。由於它可適應不同的環境氣候，因此世界各地都可見到此種植物的身影。因為其葉片的型狀，皺葉酸模又稱為長葉羊蹄。同時又被稱為酸模，因其葉片味苦的緣故。古代的中國人及羅馬人將皺葉酸模用於改善消化系統及皮膚。現在，皺葉酸模被廣泛的運用，包括改善膚質、做為輕瀉劑以及緩解肝臟問題。皺葉酸模也有助維護消化系統功能。它還有提振食慾、增加膽汁流量及強化消化等功能。與其它的草藥輕瀉劑，如蘆薈和番瀉葉一樣，皺葉酸模也含有蒽醌苷這種可促進排便的輕瀉元素。皺葉酸模還含有單寧酸，此種具消化苦性的物質能刺激食慾及膽汁流量，幫助毒素代謝，改善消化不良的症狀。

牛蒡根(Burdock Root)

牛蒡是一種備受推崇的藥用植物。它有助關節及消化系統的健康。牛蒡含有大量的菊糖及黏液，可明顯改善腸胃道不適的症狀。此外，牛蒡還有改善肝功能的作用。牛蒡也被廣泛的運用在改善乾燥粗糙的問題皮膚上。

甘草根(Licorice Root)

甘草根長期以來便有藥用及食用等兩個功能。中醫幾乎將甘草搭配在所有的藥方中。甘草最著名的使用方式，為其用於消化道及尿道的鎮痛功能（舒緩、形成保護膜）。

伏牛花樹皮及根(Barberry Bark and Root)

小檗屬伏牛花由於具有醫療上的特性，因此在全球皆受到廣泛的運用。伏牛花的根部及樹皮可分離出小蘗鹼，小蘗鹼是一種植物鹼，多個世紀以來都被運用在印度阿育吠陀醫學及中國的醫療用途上。小蘗鹼具有抑制多種微生物生長的特性。小蘗鹼也是增強免疫的草本植物金印草（白毛茛(Hydrastis canadensis)）的關鍵組成成分。小蘗鹼可以防止細菌附著在人體細胞上，因此有助促進健康的免疫系統。這種複合物也支援健康的腸胃蠕動，促進排便。小蘗胺是另一種小蘗植物內含的植物鹼，是一種抗氧化物。伏牛花內的苦性複合物，包括前面提到的植物鹼，都有助於消化。此外，伏牛花還可以支援健康的肝臟與膽囊功能。

迷迭香葉(Rosemary Leaf)

常久以來，草藥學家便將迷迭香運用在增強老年人精力和幫助消化等用途上。在中國，迷迭香亦被用來減緩頭部不適。迷迭香還有支援食慾健康、維持正常血壓、維護肝臟和膽囊健康等功用。德國的E協會（相當於美國的食品藥物管理局）甚至將迷迭香葉評定為具有治療消化不良症候群（不確定因素的消化不良）的植物。一些尚未有定論的證據提及，迷迭香或它內含的物質具有抗氧化的功效。

薑(Ginger Root)

薑是一種嫩芽狀的植物，長有紫綠色的單一花朵，土裡的粗厚根部可以食用。薑原產於印度的海邊省份，現在則在牙買加、中國，非洲和西印度群島等處種植。薑的根部是它的香氣及辣味的來源。多年來，薑被用於支援胃腸道健康並促進健康的排便。薑具有緩解暈車、暈船、噁心反胃等症狀的功效，薑同時也有益於心血管系統。

綠茶葉萃取物(Green Tea Leaf Extract)

幾個世紀以來，印度和中國皆將綠茶運用在醫療用途上。定期飲用綠茶有許多健康益處，乾燥/粉狀萃取物亦可做為膳食營養補充品。綠茶葉必須用手摘取，再經過低溫加熱以使茶葉乾燥。綠茶的活性成分有茶多酚（兒茶素）家族及黃酮醇等具高抗氧活性的物質。單寧，也就是大的多酚類分子，是綠茶裡的主要活性複合物，與和兒茶素一起組成綠茶中近90%的含量。下列有幾種兒茶素含量較高：表兒茶素(EC)、表沒食子兒茶素(EGC)、表兒茶素酸脂(ECG)和表沒食子兒茶素沒食子酸酯(EGCG)。表沒食子兒茶素沒食子酸酯(EGCG)約佔總兒茶素的10%-50%，是所有兒茶素中最強效的成分，比維生素C及維生素E的抗氧化效果更強了25-100倍。研究顯示，藉由支援健康的膽固醇及三酸甘油酯值，綠茶萃取物的健康益處有助維護心血管系統。經證實，可促進能量與免疫健康。最近的研究指出，兒茶素對促進減重具重要效果。某些研究表示，綠茶因為含有咖啡因，因此也被認為具有輕微的提升發熱作用（增加卡路里的消耗）的功能。至少有一項研究顯示，綠茶萃取物提升發熱作用的能力，遠優於綠茶內含的咖啡因能提升發熱作用的能力，也就是說綠茶的這個提升發熱的特性，應該是兒茶多酚與咖啡因交互作用的結果。一項關於綠茶的發熱作用的概論認為，因為兒茶多酚具有抑制兒茶酚-O-甲基轉移酶（一種降解正腎上腺素的酵素）的功能，因此可導致正腎上腺素值上升。

牛膝草(Hyssop)（地上部分）

牛膝草，英文學名為Hyssop officinalis，長久以來被運用在宗教及醫療等範圍內。它是種高兩尺的常綠灌木叢，開深藍色的花，原本生長於地中海地區，但現今其它地區也可發現它的蹤影。牛膝草可廣泛地用於解決消化系統問題。牛膝草中的揮發油成分，被認為是此種草藥能夠通氣除腹脹的原因。它可用來舒緩輕微的腹部絞痛與消化道不適，包括胃痛及脹氣。

紅花苜蓿(Red Clover Flower)

紅花苜蓿是莢果（豆子）家族的成員，與鷹嘴豆和黃豆屬於同類。一直以來，紅花苜蓿都被認為是清潔血液的最佳草藥。紅花苜蓿萃取物因為含有大量的大豆異黃酮複合物，因此一直被用作為膳食補充品。因為這類複合物具有類似體內雌激素活動的功能，因此經證實對更年期的婦女有所助益。一項雙盲試驗發現，紅花苜蓿可支援更年期婦女的心血管健康。據稱，紅花苜蓿還可平衡情緒、支援健康的睡眠模式、降低熱潮紅，改善性衝動、維護心臟健康以及維持骨量。

薑黃萃取物(Turmeric Root Extract)

薑黃是薑家族的成員之一。它的莖可食用，也可用於為咖哩粉增添色彩及滋味；亦可做為醫療用途。傳統上，印度阿育吠答將薑黃用於增強體能、維護視力並刺激乳汁分泌。膳食營養補充品中，薑黃本身具有抗氧化作用，與其它產品一起食用，亦可產生抗氧化功能，保護並支援腸胃道健康。薑黃的主要活性複合物為生物類黃酮、薑黃素及相關的「類薑黃素」複合物，皆具有強大的抗氧化功能。作為Nutriclean的系列產品之一，薑黃對於消化不良有很好的效果。

Timeless Prescription®美安常青配方膠囊食品：

• Abe, K., et al. Green Tea with a High Catechin Content Suppresses Inflammatory Cytokine Expression in the Galactosamine-injured Rat Liver. Biomedical Research. 26(5): 187-192, 2005.
• Alaluf, S., et al. Dietary Carotenoids Contribute to Normal Human Skin Color and UV Photosensitivity. Journal of Nutrition. 132: 399-403, 2002.
• Atalay, M., et al. Anti-angiogenic Property of Edible Berry in a Model of Hemangioma. Federation of European Biochemical Societies. 544: 252-257, 2003.
• Bagchi, D. Anti-angiogenic, Antioxidant, and Anti-carcinogenic Properties of a Novel Anthocyanin-Rich Berry Extract Formula. Biochemistry (Moscow). 69(1): 75-80, 2004.
• Beppu, M., et al. Water-soluble Antioxidants Inhibit Macrophage Recognition of Oxidized Erythrocytes. Biological & Pharmaceutical Bulletin. 24(5): 575-578, 2001.
• Bettuzzi, S., et al. Chemoprevention of Human Prostate Cancer by Oral Administration of Green Tea Catechins in Volunteers with High-grade Prostate Intraepithelial Neoplasia: a Preliminary Report from a One-year Proof-of-Principle Study. Cancer Research. 66(2): 1234-1240, 2006.
• Chan, D., et al. Regulation of Procollagen Synthesis and Processing during Ascorbate-induced Extracellular Matrix Accumulation In Vitro. Biochemical Journal. 269: 175-181, 1990.
• Chao, J. and Chu, C. Effects of Ginkgo biloba Extract on Cell Proliferation and Cytotoxicity in Human Hepatocellular Carcinoma Cells. World Journal of Gastroenterology. 10(1): 37-41, 2004.
• Chao, J., et al. Effects of Ginkgo biloba Extract on Cytoprotective Factors in Rats with Duodenal Ulcer. World Journal of Gastroenterology. 10(4): 560-566, 2004.
• Craig, W. Health-promoting Properties of Common Herbs. American Journal of Clinical Nutrition. 70(3): 491S-499S, 1999.
• Davidson, J., et al. Ascorbate Differentially Regulates Elastin and Collagen Biosynthesis in Vascular Smooth Muscle Cells and Skin Fibroblasts by Pretranslational Mechanisms. Journal of Biological Chemistry. 272 (1): 345-352, 1997.
• Davis-Searles, P., et al. Milk thistle and Prostate Cancer: Differential Effects of Pure Flavonolignans from Silybum Marianum on Antiproliferative End Points in Human Prostate Carcinoma Cells. Cancer Research. 65(10): 4448- 4457, 2005.
• de Almeida Vasconcelos Fonseca, E., et al. All-trans and 9-cis Retinoic Acids, Retinol and Beta-carotene Chemopreventive Activities during the Initial Phases of Hepatocarcinogenesis Involve Distinct Actions on Glutathione S-transferase Positive Preneoplastic Lesions Remodeling and DNA Damage. Carcinogenesis. 26(11): 1940-1946, 2005.
• Dhingra, S. and Bansal, M. Hypercholesterolemia and Apolipoprotein B Expression: Regulation by Selenium Status. Lipids in Health and Disease. 4: 28, 2005.
• Ellis, G., et al. Neutrophil Superoxide Anion–generating Capacity, Endothelial Function and Oxidative Stress in Chronic Heart Failure: Effects of Short- and Long-term Vitamin C Therapy. Journal of the American College of Cardiology. 36: 1474 – 1482, 2000.
• Engelhart, M. Dietary Intake of Antioxidants and Risk of Alzheimer Disease. The Journal of the American Medical Association. 287:3223-3229, 2002.
• Garcia-Casal, M., et al. Vitamin A and Beta-carotene Can Improve Nonheme Iron Absorption from Rice, Wheat and Corn By Humans. Journal of Nutrition. 128(3): 646-650, 1998.
• Gu, M., et al. Silibinin Inhibits Ultraviolet B Radiation-induced Mitogenic and Survival Signaling, and Associated Biological Responses in SKH-1 Mouse Skin. Carcinogenesis. 26(8): 1404-1413, 2005. 
• Gu, M., et al. Silibinin Protects against Photocarcinogenesis via Modulation of Cell Cycle Regulators, Mitogen-Activated Protein Kinases, and Akt Signaling. Cancer Research. 64: 6349-6356, 2004.
• He, S., et al. Effects of Extract from Ginkgo biloba on Carbon Tetrachloride-induced Liver Injury in Rats. World Journal of Gastroenterology. 12(24): 3924-3928, 2006.
• Head, K. Natural Therapies for Ocular Disorders, Part Two: Cataracts and Glaucoma. Alternative Medicine Review. 6(2): 141-166, 2001.
• Heinrich, U., et al. Supplementation with {beta}-Carotene or a Similar Amount of Mixed Carotenoids Protects Humans from UV-Induced Erythema. Journal of Nutrition. 133(1): 98-101, 2003.
• Hsu, S. and Dickinson, D. A New Approach to Managing Oral Manifestations of Sjogren's Syndrome and Skin Manifestations of Lupus. Korean Society for Biochemistry and Molecular Biology. 39(3): 229-239, 2006.
• Huang, H., et al. Effects of Vitamin C and Vitamin E on In Vivo Lipid Peroxidation: Results of a Randomized Controlled Trial. American Journal of Clinical Nutrition. 76: 549 – 555, 2002.
• Huang, S., et al. Effect of Ginkgo biloba Extract on Livers in Aged Rats. World Journal of Gastroenterology. 11(1): 132-135, 2005.
• Ito, Y., et al. Cardiovascular Disease Mortality and Serum Carotenoid Levels: a Japanese Population-based Follow-up Study. Journal of Epidemiology. 16(4): 154-160, 2006.
• Jang, Y., et al. Anthocyanins Protect against A2E Photooxidation and Membrane Permeabilization in Retinal Pigment Epithelial Cells. Photochemistry and Photobiology. 81(3): 529-536, 2005.
• Jia, X. and Han, C. Chemoprevention of Tea on Colorectal Cancer Induced by Dimethylhydrazine in Wistar Rats. World Journal of Gastroenterology. 6(5): 699-703, 2000.
• Kidd, P. A Review of Nutrients and Botanicals in the Integrative Management of Cognitive Dysfunction. Alternative Medicine Review. 4(3): 144-161, 1999.
• Kim, H., et al. Effects of Green Tea Polyphenol on Cognitive and Acetylcholinesterase Activities. Bioscience, Biotechnology, and Biochemistry. 68(9): 1977-1979, 2004.
• Kren, V. and Walterova, D. Silybin and Silymarin—New Effects and Applications. Biomedical Papers. 149(1): 29-41, 2005.
• Kupka, R., et al. Selenium Status is Associated with Accelerated HIV Disease Progression among HIV-1-infected Pregnant Women in Tanzania. Journal of Nutrition. 134: 2556-2560, 2004.
• Lasse, M., et al. Anthocyanins Induce Cell Cycle Perturbations and Apoptosis in Different Human Cell Lines. Carcinogenesis. 25(8): 1427-1433, 2004.
• Li, L., et al. Silibinin Prevents UV-Induced HaCaT Cell Apoptosis Partly through Inhibition of Caspase-8 Pathway. Biological & Pharmaceutical Bulletin. 29(6): 1096-1101, 2006.
• Logan, A. and Wong, C. Chronic Fatigue Syndrome: Oxidative Stress and Dietary Modifications. Alternative Medicine Review. 6(5): 450-459, 2001.
• Manna, S. Silymarin Suppresses TNF-induced Activation of NF-kappa B, c-Jun N-terminal Kinase, and Apoptosis. Journal of Immunology. 163(12): 6800-6809, 1999.
• Mantena, S., et al. Orally Administered Green Tea Polyphenols Prevent Ultraviolet Radiation-Induced Skin Cancer in Mice through Activation of Cytotoxic T Cells and Inhibition of Angiogenesis in Tumors. Journal of Nutrition. 135: 2871-2877, 2005.
• Mayne, S. Antioxidant Nutrients and Chronic Disease: Use of Biomarkers of Exposure and Oxidative Stress Status in Epidemiologic Research. Journal of Nutrition. 133: 933S-940S, 2003.
• Mayne, S. Antioxidant Nutrients and Chronic Disease: Use of Biomarkers of Exposure and Oxidative Stress Status in Epidemiologic Research. Journal of Nutrition. 133: 933S-940S, 2003.
• McArdle, F., et al. Effects of Oral Vitamin E and ß-carotene Supplementation on Ultraviolet Radiation–induced Oxidative Stress in Human Skin. American Journal of Clinical Nutrition. 80(5): 1270-1275, 2004.
• Nakajima, J., et al. LC/PDA/ESI-MS Profiling and Radical Scavenging Activity of Anthocyanins in Various Berries. Journal of Biomedicine and Biotechnology. 2004(5): 241-247, 2004.
• Ogasawara, M., et al. Differential Effects of Antioxidants on the In Vitro Invasion, Growth and Lung Metastasis of Murine Colon Cancer Cells. Biological & Pharmaceutical Bulletin. 30(1):200-204, 2007.
• Ohta, Y., et al. Effect of Oral Vitamin E Administration on Acute Gastric Mucosal Lesion Progression in Rats Treated with Compound 48/80, a Mast Cell Degranulator. Biological & Pharmaceutical Bulletin. 29 (4):675-683, 2006.
• Paleologos, M., et al. Cohort Study of Vitamin C Intake and Cognitive Impairment. American Journal of Epidemiology. 148(1):45-50, 1998.
• Park, Y., et al. Preventive Effect of Ginkgo biloba Extract (GBB) on the Lipopolysaccharide-induced Expressions of Inducible Nitric Oxide Synthase and Cyclooxygenase-2 via Suppression of Nuclear Factor-kappaB in RAW 264.7 Cells. Biological & Pharmaceutical Bulletin. 29(5): 985-990, 2006.
• Pence, B., et al. Effects of Dietary Selenium on UVB-Induced Skin Carcinogenesis and Epidermal Antioxidant Status. Journal of Investigative Dermatology. 102: 759-761, 1994.
• Placzek, M., et al. Ultraviolet B-Induced DNA Damage in Human Epidermis Is Modified by the Antioxidants Ascorbic Acid and D-alpha-Tocopherol. Journal of Investigative Dermatology. 124: 304-307, 2005.
• Ravindranath, M., et al. Epicatechins Purified from Green Tea (Camellia Sinensis) Differentially Suppress Growth of Gender-Dependent Human Cancer Cell Lines. Evidence Based Complementary and Alternative Medicine. 3(2): 237-247, 2006.
• Ruggiero, P., et al. Red Wine and Green Tea Reduce H pylori- or VacA-induced Gastritis in a Mouse Model. World Journal of Gastroenterology. 13(3): 349-354, 2007.
• Salonen, R., et al. Six-Year Effect of Combined Vitamin C and E Supplementation on Atherosclerotic Progression: The Antioxidant Supplementation in Atherosclerosis Prevention (ASAP) Study. Circulation. 107: 947 – 953, 2003.
• Sato, Y., et al. The Feeding of ?-Carotene Down-Regulates Serum IgE Levels and Inhibits the Type I Allergic Response in Mice. Biological & Pharmaceutical Bulletin. 27(7): 978-984, 2004.
• Shen, X., et al. Effects of Dietary Supplementation with Vitamin E and Selenium on Rat Hepatic Stellate Cell Apoptosis. World Journal of Gastroenterology. 11(32): 4957-4961, 2005.
• Simon, J., et al. Relation of Serum Ascorbic Acid to Mortality among US Adults. Journal of the American College of Nutrition. 20: 255-263, 2001.
• Singh, R., et al. Dietary feeding of Silibinin Inhibits Advance Human Prostate Carcinoma Growth in Athymic Nude Mice and Increases Plasma Insulin-like Growth Factor-binding Protein-3 Levels. Cancer Research. 62(11): 3063-3069, 2002.
• Sparrow, J., et al. A2E-epoxides Damage DNA in Retinal Pigment Epithelial Cells. Vitamin E and Other Antioxidants Inhibit A2E-epoxide Formation. Journal of Biological Chemistry. 278(20): 18207-18213, 2003.
• Trevithick-Sutton, C., et al. The Retinal Carotenoids Zeaxanthin and Lutein Scavenge Superoxide and Hydroxyl Radicals: A ChemilumInescence and ESR Study. Molecular Vision. 12: 1127-1135, 2006.
• Tyagi, A., et al. Silibinin Strongly Synergizes Human Prostate Carcinoma DU145 Cells to Doxorubicin-induced Growth Inhibition, G2-M Arrest, and Apoptosis. Clinical Cancer Research. 8(11): 3512-3119, 2002.
• Vahlquist, A., et al. Vitamin A in Human Skin: II Concentrations of Carotene, Retinol and Dehydroretinol in Various Components of Normal Skin. Journal of Investigative Dermatology. 79: 94-97, 1982.
• van der Brandt, P., et al. Toenail Selenium Levels and the Subsequent Risk of Prostate Cancer: a Prospective Cohort Study. Cancer Epidemiology Biomarkers & Prevention. 12: 866-871, 2003.
• van Rooij, J., et al. Oral Vitamins C and E as Additional Treatment in Patients with Acute Anterior Uveitis: a Randomised Double Masked Study in 145 Patients. British Journal of Ophthalmology. 83: 1277-1282, 1999.
• Varghese, L., et al. Silibinin Efficacy against Human Hepatocellular Carcinoma. Clinical Cancer Research. 11: 8441-8448, 2005.
• Wang, Z., et al. Inhibitory Effects of Black Tea, Green Tea, Decaffeinated Black Tea, and Decaffeinated Green Tea on Ultraviolet B Light-induced Skin Carcinogenesis in 7,12-Dimethylbenz[a]anthracene-initiated SKH-1 Mice. Cancer Research. 54(13): 3428-3435, 1994.
• Wei, W., et al. Prospective Study of Serum Selenium Concentrations and Esophageal and Gastric Cardia Cancer, Heart Disease, Stroke, and Total Death. American Journal of Clinical Nutrition. 79(1): 80-85, 2004.
• Wertz, K., et al. -Carotene Interferes with Ultraviolet Light A-Induced Gene Expression by Multiple Pathways. Journal of Investigative Dermatology. 124: 428-434, 2005.
• Xu, A., et al. Therapeutic Mechanism of Ginkgo biloba Exocarp Polysaccharides on Gastric Cancer. World Journal of Gastroenterology. 9(11): 2424-2427, 2003.
• Yoshida, M., et al. Combined Effect of Vitamin E and Insulin on Cataracts of Diabetic Rats Fed a High Cholesterol Diet. Biological & Pharmaceutical Bulletin. 27(3): 338-344, 2004.
• You, W. Gastric Dysplasia and Gastric Cancer: Helicobacter pylori, Serum Vitamin C, and Other Risk Factors. Journal of the National Cancer Institute. 92(19): 1607-1612, 2000.
• Zhang, X., et al. Effect of Tea Polyphenol on Cytokine Gene Expression in Rats with Alcoholic Liver Disease. Hepatobiliary & Pancreatic Disease International. 5(2): 268-272, 2006.
• Zhou, B., et al. Silibinin Protects Rat Cardiac Myocyte from Isoproterenol-Induced DNA Damage Independent on Regulation of Cell Cycle. Biological & Pharmaceutical Bulletin. 29(9): 1900-1905, 2006.
• Zigman, S. Effects of Green Tea Polyphenols on Lens Photooxidative Stress. Biological Bulletin. 197: 285-286, 1999.

Curcumin Extreme™薑黃素配方：

• Araujo, C. and Leon, L. Biological activities of Curcuma longa L. Memorias do Instituto Oswaldo Cruz. 96(5): 723-728, 2001.
• Bhattacharyya, S., et al. Curcumin prevents tumor-induced T cell apoptosis through Stat-5a-mediated Bcl-2 induction. Journal of Biological Chemistry. 282(22): 15954-15964.
• Biswas, S., et al. Curcumin induces glutathione biosynthesis and inhibits NF-kappaB activation and interleukin-8 release in alveolar epithelial cells: mechanism of free radical scavenging activity. Antioxidants and Redox Signaling. 7(1-2): 32-41, 2005.
• Cheng, Y., et al. Effects of curcumin on peroxisome proliferator-activated receptor gamma expression and nuclear translocation/redistribution in culture-activated rat hepatic stellate cells. Chinese Medical Journal. 120(9): 794-801, 2007.
• Churchill, M., et al. Inhibition of intestinal tumors by curcumin is associated with changes in the intestinal immune cell profile. Journal of Surgical Research. 89(2): 169-175, 2000.
• Cornblatt, B., et al. Preclinical and clinical evaluation of sulforaphane for chemoprevention in the breast. 28(7): 1485-1490, 2007.
• Dairam, A., et al. Curcuminoids, curcumin, and demethoxycurcumin reduce lead-induced memory deficits in male Wistar rats. Journal of Agricultural and Food Chemistry. 55(3): 1039-1044, 2007.
• Dickinson, D., et al. Curcumin alters EpRE and AP-1 binding complexes and elevates glutamate-cysteine ligase gene expression. FASEB. 17(3): 473-475, 2003.
• Fahey, J., et al. Sulforaphane inhibits extracellular, intracellular, and antibiotic-resistant strains of Helicobacter pylori and prevents benzo[a]pyrene-induced stomach tumors. Proceedings of the National Academy of Sciences of the United States of America. 99(11): 7610-7615, 2002.
• Farombi, E., et al. Curcumin attenuates dimethylnitrosamine-induced liver injury in rats through Nrf2-mediated induction of heme oxygenase-1. Food and Chemical Toxicology. 46(4): 1279-1287, 2008.
• Funk, J., et al. Turmeric extracts containing curcuminoids prevent experimental rheumatoid arthritis. Journal of Natural Products. 69(3): 351-355, 2006.
• Gao, X. and Talalay, P. Induction of phase 2 genes by sulforaphane protects retinal pigment epithelial cells against photooxidative damage. Proceedings of the National Academy of Sciences of the United States of America. 101(28): 10446-10451, 2004.
• Garcia-Alloza, M., et al. Curcumin labels amyloid pathology in vivo, disrupts existing plaques, and partially restores distorted neurites in an Alzheimer mouse model. Journal of Neurochemistry. 102(4): 1095-1104, 2007.
• Higdon, J., et al. Cruciferous vegetables and human cancer risk: epidemiologic evidence and mechanistic basis. Pharmacological Research. 55(3): 224-236, 2007.
• Howells, L., et al. Comparison of oxaliplatin- and curcumin-mediated antiproliferative effects in colorectal cell lines. International Journal of Cancer. 121(1): 175-183, 2007.
• Jagetia, G. and Aggarwal, B. "Spicing up" of the immune system by curcumin. Journal of Clinical Immunology. 27(1): 19-35, 2007.
• Johnson, J., et al. Curcumin for chemoprevention of colon cancer. Cancer Letters. 255(2): 170-181, 2007.
• Juge, N., et al. Molecular basis for chemoprevention by sulforaphane: a comprehensive review. Cellular and Molecular Life Sciences. 64(9): 1105-1127, 2007.
• Kaur, G., et al. Inhibition of oxidative stress and cytokine activity by curcumin in amelioration of endotoxin-induced experimental hepatoxicity in rodents. Clinical and Experimental Immunology. 145(2): 313-321, 2006.
• Kim, G., et al. Curcumin inhibits immunostimulatory function of dendritic cells: MAPKs and translocation of NF-kappa B as potential targets. Journal of Immunology. 174(12): 8116-8124, 2005.
• Kurup, V., et al. Immune response modulation by curcumin in a latex allergy model. Clinical and Molecular Allergy. 5: 1, 2007.
• Lim, G., et al. The curry spice curcumin reduces oxidative damage and amyloid pathology in an Alzheimer transgenic mouse. Journal of Neuroscience. 21(21): 8370-8377, 2001.
• Lin, J. Molecular targets of curcumin. Advances in Experimental Medicine and Biology. 595: 227-243, 2007.
• Magalska, A., et al. Curcumin induces cell death without oligonucleosomal DNA fragmentation in quiescent and proliferating human CD8+ cells. Acta Biochimica Polonica. 53(3): 531-538, 2006.
• Maheshwari, R., et al. Multiple biological activities of curcumin: a short review. Life Sciences. 78(18): 2081-2087, 2006.
• Mathuria, N. and Verma, R. Ameliorative effect of curcumin on aflatoxin-induced toxicity in DNA, RNA and protein in liver and kidney of mice. Acta Poloniae Pharmaceutica. 64(6): 497-502, 2007.
• Monograph. Curcuma longa (turmeric). Alternative Medicine Review. 6(suppl): S62-S66, 2001.
• Morimitsu, Y., et al. A sulforaphane analogue that potently activates the Nrf2-dependent detoxification pathway. Journal of Biological Chemistry. 277(5): 3456-3463, 2002.
• Myzak, M. and Dashwood, R. Chemoprotection by sulforaphane: keep one eye beyond Keap1. Cancer Letters. 233(2): 208-218, 2006.
• Myzak, M., et al. Sulforaphane inhibits histone deacetylase in vivo and suppresses tumorigenesis in Apc-minus mice. FASEB. 20(3): 506-508, 2006.
• Naik, R., et al. Protection of liver cells from ethanol cytotoxicity by curcumin in liver slice culture in vitro. Journal of Ethnopharmacology. 95(1): 31-37, 2004.
• Nanji, A., et al. Curcumin prevents alcohol-induced liver disease in rats by inhibiting the expression of NF-kappa B-dependent genes. American Journal of Physiology. 284(2): G321-G327, 2003.
• Ng, T., et al. Curry consumption and cognitive function in the elderly. American Journal of Epidemiology. 164(9): 898-906, 2006.
• Nishinaka, T., et al. Curcumin activates human glutathione S-transferase P1 expression through antioxidant response element. Toxicology Letters. 170(3): 238-247, 2007.
• Noyan-Ashraf, M., et al. Dietary approach to decrease aging-related CNS inflammation. Nutritional Neuroscience. 8(2): 101-110, 2005.
• O’Connell, M. and Rushworth, S. Curcumin: potential for hepatic fibrosis therapy? British Journal of Pharmacology. 153(3): 403-405, 2007.
• Osawa, T. Nephroprotective and hepatoprotective effects of curcuminoids. Advances in Experimental Medicine and Biology. 595: 407-423, 2007.
• Pal, S., et al. Amelioration of immune cell number depletion and potentiation of depressed detoxification system of tumor-bearing mice by curcumin. Cancer Detection and Prevention. 29(5): 470-478, 2005.
• Pari, L. and Amali, D. Protective role of tetrahydrocurcumin (THC) an active principle of turmeric on chloroquine induced hepatotoxicity in rats. Journal of Pharmacy and Pharmaceutical Sciences. 8(1): 115-123, 2005.
• Perkins, S., et al. Chemopreventive efficacy and pharmacokinetics of curcumin in the min/+ mouse, a model of familial adenomatous polyposis. Cancer Epidemiology, Biomarkers, and Prevention. 11(6): 535-540, 2002.
• Rushworth, S., et al. Role of protein kinase C delta in curcumin-induced antioxidant response element-mediated gene expression in human monocytes. Biochemical and Biophysical Research Communications. 341(4): 1007-1016, 2006.
• Salvioli, S., et al. Curcumin in cell death processes: A challenge for CAM of age-related pathologies. Evidence-based Complementary and Alternative Medicine. 4(2): 181-190, 2007.
• Scapagnini, G., et al. Curcumin activates defensive genes and protects neurons against oxidative stress. Antioxidants and Redox Signaling. 8(3-4): 395-403, 2006.
• Shen, G., et al. Modulation of nuclear factor E2-related factor 2-mediated gene expression in mice liver and small intestine by cancer chemopreventive agent curcumin. Molecular and Cancer Therapeutics. 5(1): 39-51, 2006.
• Shen, S., et al. Protective effect of curcumin against liver warm ischemia/reperfusion injury in rat model is associated with regulation of heat shock protein and antioxidant enzymes. World Journal of Gastroenterology. 13(13): 1953-1961, 2007.
• Shishodia, S., et al. Curcumin: getting back to the roots. Annals of the New York Academy of Sciences. 1056: 206-217, 2005.
• Shu, J., et al. The study of therapeutic effects of curcumin on hepatic fibrosis and variation of correlated cytokine. Journal of Chinese Medicinal Materials. 30(11): 1421-1425, 2007.
• Shu, J., et al. Therapeutic effects of curcumin treatment on hepatic fibrosis. Chinese Journal of Hepatology. 15(10): 753-757, 2007.
• Shukla, P., et al. Protective effect of curcumin against lead neurotoxicity in rat. Human and Experimental Toxicology. 22(12): 653-658, 2003.
• Smith, T., et al. Allyl-isothiocyanate causes mitotic block, loss of cell adhesion and disrupted cytoskeletal structure in HT29 cells. Carcinogenesis. 25(8): 1409-1415, 2004.
• Srinivasan, M., et al. Protective effect of curcumin on gamma-radiation induced DNA damage and lipid peroxidation in cultured human lymphocytes. Mutation Research. 611(1-2): 96-103, 2006.
• Tang, L., et al. Potent activation of mitochondria-mediated apoptosis and arrest in S and M phases of cancer cells by a broccoli sprout extract. Molecular Cancer Therapeutics. 5(4): 935-944, 2006.
• Thangapazham, R., et al. Multiple molecular targets in cancer chemoprevention by curcumin. AAPS Journal. 8(3): E443-E449, 2006.
• Thejass, P. and Kuttan, G. Antimetastatic activity of Sulforaphane. Life Sciences. 78(26): 3043-3050, 2006.
• Thejass, P. and Kuttan, G. Augmentation of natural killer cell and antibody-dependent cellular cytotoxicity in BALB/c mice by sulforaphane, a naturally occurring isothiocyanate from broccoli through enhanced production of cytokines IL-2 and IFN-gamma. Immunopharmacology and Immunotoxicology. 28(3): 443-457, 2006.
• Thejass, P. and Kuttan, G. Immunomodulatory activity of Sulforaphane, a naturally occurring isothiocyanate from broccoli (Brassica oleracea). Phytomedicine. 14(7-8): 538-545, 2007.
• Wakabayashi, N., et al. Protection against electrophile and oxidant stress by induction of the phase 2 response: fate of cysteines of the Keap1 sensor modified by inducers. Proceedings of the National Academy of Sciences of the United States of America. 101(7): 2040-2045, 2004.
• Wei, Q., et al. Inhibition of lipid peroxidation and protein oxidation in rat liver mitochondria by curcumin and its analogues. Biochimica et Biophysica Acta. 1760(1): 70-77, 2006.
• Wu, A., et al. Dietary curcumin counteracts the outcome of traumatic brain injury on oxidative stress, synaptic plasticity, and cognition. 197(2): 309-317, 2006.
• Xu, Y., et al. Curcumin reverses the effects of chronic stress on behavior, the HPA axis, BDNF expression and phosphorylation of CREB. Brain Research. 1122(1): 56-64, 2006.
• Yadav, V., et al. Immunomodulatory effects of curcumin. Immunopharmacology and Immunotoxicology. 27(3): 485-497, 2005.
• Yang, F., et al. Curcumin inhibits formation of amyloid beta oligomers and fibrils, binds plaques, and reduces amyloid in vivo. Journal of Biological Chemistry. 280(7): 5892-5901, 2005.
• Ye, S., et al. Effect of curcumin on the induction of glutathione S-transferases and NADP(H):quinone oxidoreductase and its possible mechanism of action. Acta Pharmaceutica Sinica. 42(4): 376-380, 2007.
• Zhang, L., et al. Curcuminoids enhance amyloid-beta uptake by macrophages of Alzheimer's disease patients. Journal of Alzheimer’s Disease. 10(1): 1-7, 2006.
• Zheng, S. and Chen, A. Curcumin suppresses the expression of extracellular matrix genes in activated hepatic stellate cells by inhibiting gene expression of connective tissue growth factor. American Journal of Physiology. 290(5): G883-G893, 2006.
• Zheng, S. and Chen, A. Disruption of transforming growth factor-beta signaling by curcumin induces gene expression of peroxisome proliferator-activated receptor-gamma in rat hepatic stellate cells. American Journal of Physiology. 292(1): G113-G123, 2007.
• Zheng, S., et al. De novo synthesis of glutathione is a prerequisite for curcumin to inhibit hepatic stellate cell (HSC) activation. Free Radical Biology and Medicine. 43(3): 444-453, 2007.

NutriClean®清肝配方：

• Varghese, L., et al. Silibinin Efficacy against Human Hepatocellular Carcinoma. Clinical Cancer Research. 11: 8441-8448, 2005.
• Rainone, F. Milk Thistle. American Family Physician. 72(7): 1285-1288,2005.
• Kidd, P. and Head, K. A Review of the Bioavailability and Clinical Efficacy of Milk Thistle Phytosome: a Silybin-Phosphatidylcholine Complex (Siliphos). . Alternative Medicine Review. 10(3): 193-203, 2005.
• Luper, S. A Review of Plants Used in the Treatment of Liver Disease: Part Two. Alternative Medicine Review. 4(3): 178-188, 1999.
• Cetinkaya, A., et al. N-Acetylcysteine Ameliorates Methotrexate-Induced Oxidative Liver Damage in Rats. Medical Science Monitor. 12(8): 274-278, 2006.
• Pal, R., et al. Effect of Garlic on Isoniazid and Rifampicin-Induced Hepatic Injury in Rats. World Journal of Gastroenterology. 12(4); 636-639, 2006.
• Kidd, P. and Head, K. A Review of the Bioavailability and Clinical Efficacy of Milk Thistle Phytosome: a Silybin-Phosphatidylcholine Complex (Siliphos). Alternative Medical Review. 10(3): 193-203, 2005.
• Luper, S. A Review of Plants Used in the Treatment of Liver Disease: Part One. Alternative Medicine Review. 3(6): 410-421, 1998.
• Pal, R., et al. Effect of Garlic on Isoniazid and Rifampicin-Induced Hepatic Injury in Rats. World Journal of Gastroenterology. 12(4); 636-639, 2006.
• Cetinkaya, A., et al. N-Acetylcysteine Ameliorates Methotrexate-Induced Oxidative Liver Damage in Rats. Medical Science Monitor. 12(8): 274-278, 2006.
• Pal, R., et al. Effect of Garlic on Isoniazid and Rifampicin-Induced Hepatic Injury in Rats. World Journal of Gastroenterology. 12(4); 636-639, 2006.
• Kren, V. and Walterova, D. Silybin and Silymarin--New Effects and Applications. Biomedical papers of the Medical Faculty of the University Palack.